Age: 41Yrs
Sex: Female
Case Study: Well-defined T2/STIR hyperintense signal cystic lesion in the postero-lateral aspect of vagina.
Images:
Conclusion : Postero-lateral wall of vagina shows a cyst as described – Suggestive of likely Bartholin’s cyst.
Age: 11 Yrs
Sex: Female
Complaints: Developmental abnormalities. K/c/o HIE.
Case study: Evidence of periventricular T2/FLAIR hyperintensities seen in the posterior fossa around the occipital horn extending into the adjacent sulcal spaces. The corpus callosum shows significant thinning predominantly involving the isthmus and splenium. Volume loss seen in the right hippocampal head with widening of CSF space. There is asymmetric ex-vacuo dilatation of right occipital horn of lateral ventricle.
Images:
Conclusion: Atrophic changes involving the bilateral cerebral hemisphere in the form of periventricular and deep white matter hyperintensities showing cortical thinning along with asymmetric ex-vacuo dilatation of occipital horn. Corpus callosal thinning involving the isthmus and splenium. Right hippocampal volume loss. – Features are in consistent with atrophic changes as sequelae of Hypoxic Ischemic Encephalopathy.
Age: 48yr
Sex: Female
Complaints: Sudden headache and unconsciousness.
Case study: Extra axial, crescent shaped, hyperdense areas noted along the right temporo-fronto-parietal convexity (maximum thickness ~22 mm) causing mass effect in the form of effacement of ipsilateral sulcal spaces, compression of the ipsilateral lateral ventricle, subfalcine hernia and midline shift (~ 11mm) to the left.
Images:
Conclusion: Subdural hemorrhage along the right tempero-fronto-parietal convexity causing mass effect and midline shift.
Sex: Male
Complaints: Left Flank pain.
Age: 48yr
Case study: Left kidney: 10.8 X 3.6 cm. Minimal perinephric free fluid with fat stranding noted. A calculus (~HU 581) measuring ~ 7.7 x 5.7 x 5.4 mm (CC x AP x TR)noted in the upper segment of left ureter with periureteric fat stranding through out its course.
Images:
On contrast administration left kidney in delayed phase:
4 min delay image shows : No contrast transport in the ureter.
10 min delay image shows: No contrast transport in the ureter. There is evidence of contrast extravasation tracing from the renal hila extending circumferentially around proximal ureter confined to the upper segment.
20 min delay image shows: No contrast transport in the ureter. There is evidence of contrast extravasation tracing from the renal hila and mid calyx extending into the perinephric and periureteric region. There is irregular streaking of contrast extending into the adjacent perinephric plane both medially and laterally.
30 min delay images shows: No contrast transport in the left ureter. There is further more extravasation of contrast along the ureter caudally in addition to previously seen mentioned areas.
Conclusion: Left ureteric calculus with renal inflammatory changes in the form of perinephric fluid and fat stranding. Left kidney shows contrast leakage tracing from the hila as described in delayed phase with increase in area of spread proportionate to increase in time. – ?pelvic/calyceal rupture. Left ureter shows no contrast transport through out the multiple phase imaging study. No urinary bladder calculus.
Age: 28yr
Sex: Male
Complaints: Progressive walking difficulty.
Case study: T2 hypointensity noted in the pons and midbrain region with mild reduction in the bulk of the vermis. Mild prominence of the cisterna magna noted. The cerebellar foliae appears mildly prominent. Long TR hyperintensities noted in the white matter regions of the corona radiata and centrum semiovale with no abnormality on the T1WI.
Images:
PERFUSION CLUT VIEW
T2WI Enlarged view of pons showing linear hypointensities.
T2WI Heavy Weighted sequence shows prominent cisterna magna.
FLAIR shows hypointense linear areas on pons.
T2WI shows hyperintense scattered and confluent areas involving corona radiata and centrum semiovale.
Conclusion : K/c/o spastic ataxia and progressive walking difficulty follow up;T2 hypointensities in the region of the midbrain and pons with reduced bulk of the vermis and prominence of the cerebellar foliae.
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